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These changes may seem simple, but together they contribute to a stronger foundation for long-term success

Structures were solved by molecular replacement using Phaser-MR and chain A of PDB 6U32 as the model

Standard subcutaneous reconstitution

Our spatial transcriptomics data on 12- and 17-week-old untreated NOD mice confirmed progressive upregulation of ER stress and UPR genes, downregulation of beta cell identity markers and enhanced antigen presentation and proinflammatory signalling